www.aqll-lab.cl

The main research topic focusses on studing the molecular basis of intercellular communication that cells use to respond to cues provided by the surrounding microenvironment. Our team, of 20-25 members, is a dynamic group studying several aspects of cell signaling. Our interests include regulation of cell death, molecular pathways controlling cell transformation, as well as the mechanisms involved in communication between cells of the central nervous system.

Our lab is part of the FONDAP Center for Molecular Studies of the Cell and the Biomedical Neuroscience Institute of the Institute of Biomedical Science of the Faculty of Medicine, University of Chile. As part of these groups, we are committed to research, post-graduate teaching and training of undergraduate as well as postgraduate students.

Our Team

Dr. Andrew Quest

Is a Biochemist from the ETH, Zürich, Switzerland that got his PhD in Natural Sciences in the ETH, Zürich, Switzerland and is currently a Full Professor at the Faculty of Medicine, University of Chile. He is also the deputy Director of the FONDAP Center for Molecular Studies of the Cell (CEMC).

This e-mail address is being protected from spambots. You need JavaScript enabled to view it

Research: Signal transduction focused on elucidating the molecular mechanisms employed by Caveolin-1 to modulate tumour cell behavior. His lab also studies different mechanisms of cell death triggered by activation of the Fas receptor.

Dr. Lisette Leyton

Is a Biochemist from the Faculty of Chemical and Pharmaceutical Sciences, University of Chile that got her PhD in Sciences in the Faculty of Basic Sciences, University of Chile. She is currently an Associate Professor at the Faculty of Medicine, University of Chile and also part of the Committee of the Postgraduate Program in Biomedical Sciences of the same Faculty.

This e-mail address is being protected from spambots. You need JavaScript enabled to view it

Research: Signal transduction events triggered by neuron-astrocyte interactions, their role in astrocyte activation (morphological changes and migration) and neuronal cell death in response to pro-inflammatory stimuli.